Removed at Facebook, my discussions regarding the RaTG13

Latham and Wilson say: "An intriguing alternative possibility is that SARS-CoV-2 acquired its furin site directly from the miner’s lungs (since 2012!!!). Humans possess an epithelial sodium channel protein called ENaC-a whose furin cleavage site is identical over eight amino acids to SARS-CoV-2 (look at:

Article SARS-CoV-2 strategically mimics proteolytic activation of human ENaC

)... SARS-CoV-2 is highly adapted across its whole set of component proteins and not just at the spike ...It is hard to imagine an explanation for this high adaptiveness other than some kind of passaging in a human body ... the spike and the RBD evolved in tandem and in the presence of the human immune system (i.e. in a human body and not in tissue culture; their notes, the previous and this amazing one, the "hit-piece" is pointing at the passage through humans!!!, The 2012 case of the Mojiang miners!!!: "such features suggest the involvement of an immune system” (Andersen et al., 2020). )". "We refer to this COVID-19 origin hypothesis as the Mojiang Miners Passage (MMP) hypothesis... According to the Master’s thesis, samples were taken from patients for “scientific research” and blood samples (at least) were sent to the WIV... The MMP proposal has the additional benefit of reconciling many observations concerning SARS-CoV-2 that have proven difficult to reconcile with any natural zoonotic hypothesis... our MMP proposal requires only the one species jump, which is documented in the Master’s thesis... the MMP theory (my note: That COVID-19 was in the hands of Zheng-Li since 2012, but she deliberately did not disclose it, making a lot of unethical moves as a scientist: Changing names, hiding relevant data and references, concealing key details, etc., etc., which only raises more and more questions pointing at her total dishonesty as a scientist, and her total commitment to the CCP as another of its disposable "pawns" such as: "Why in the title of the Ge et al. 2016 paper did the Shi lab call it an “abandoned” mine? (if miners were working there and even died by a SARS related, a.k.a., COVID-19 viral infection!!!!)"!!!) is a plausible and parsimonious explanation of all the key features of the COVID-19 pandemic and its origin. It accounts for 1) the propensity of SARS-CoV-2 infections to target the lungs; 2) the apparent preadapted nature of the virus; and 3) its transmission from bats in Yunnan to humans in Wuhan."

Furthermore the full original MSc Thesis of the first 2013 COVID-19-likes is available there for download: https://assets.documentcloud.org/documents/6981198/Analysis-of-Six-Patients-With-Unknown-Viruses.pdf

As they describe the 2013 symptoms of that COVID-19-like scenario (that Shi Zheng-Li has tried to keep on hidden with every single trick in her book: changing names of the strain, omitting that it was a virus then the cause, etc.): "a) dry cough, b) sputum, c) high fevers, especially shortly before death d) difficulty breathing, e) myalgia (sore limbs). Some patients had hiccoughs and headaches... Clinical work established that patients 1-4 had low blood oxygen “for sure it was ARDS” (Acute Respiratory Distress Syndrome) and immune damage considered indicative of viral infection. Additionally, a tendency for thrombosis was noted in patients 2 and 4. Symptom severity and mortality were age-related... Treatment of the six patients included ventilation (patients 2-4), steroids (all patients), antivirals (all except patient 5), and blood thinners (patients 2 and 4). Antibiotics and antifungal medications were administered to counter what were considered secondary (but significant) co-infections... Judging by their clinical symptoms such as the CT scans, all the miner’s infections were primarily of the lungs... the hospitalised miners had very long-term bouts of disease characterised by a continuous high load of virus. In the cases of patients 3 and 4 their illnesses lasted over 4 months..." and most importantly, as per our HIV INSERTIONS (in the word used by Zheng-Li herself!!!): "Furthermore, according to the Master’s thesis, the immune systems of the miners were compromised and remained so even for those discharged"!!! Whoaaaa!!! And also: "Coronaviruses are well known to recombine at very high rates: 10% of all progeny in a cell can be recombinants" Now my question is: Can a coronavirus even pick up in a host a favorable sequence from bacteria????

Regarding multiple "passages" within the lungs of these miners, this is what they say: "...movement around the lungs would likely have resulted in what amounted to a passaging effect without the need for a researcher to infect new tissues. Indeed, in the Master’s thesis the observation is several times made that areas of the lungs of a specific patient would appear to heal even while other parts of the lungs would become infected." Just like today!!!

This is their straightforward conclusion: "We suggest, first, that inside the miners RaTG13 (or a very similar virus) evolved into SARS-CoV-2, an unusually pathogenic coronavirus highly adapted to humans. Second, that the Shi lab used medical samples taken from the miners and sent to them by Kunming University Hospital for their research. It was this human-adapted virus, now known as SARS-CoV-2, that escaped from the WIV in 2019. / We refer to this COVID-19 origin hypothesis as the Mojiang Miners Passage (MMP) hypothesis."

Their previous article is: https://archive.vn/RxbFv

Plus the classic statement by, RIP, Mullis, about the uselessness of PCR for estimating viral load: "With regard to the viral-load tests, which attempt to use PCR for counting viruses, Mullis has stated: “Quantitative PCR is an oxymoron.” PCR is intended to identify substances qualitatively, but by its very nature is unsuited for estimating numbers. Although there is a common misimpression that the viral-load tests actually count the number of viruses in the blood, these tests cannot detect free, infectious viruses at all; they can only detect proteins that are believed, in some cases wrongly, to be unique to HIV. The tests can detect genetic sequences of viruses, but not viruses themselves." From: Lauritsen J. Has Provincetown Become Protease Town? [Internet]. Available from: http://www.virusmyth.org/aids/hiv/jlprotease.htm

Here is the preprint also mentioned in that article of the Preprint of Segreto and now Deigin!!!!

Finally this is the link, posted elsewhere, that was removed from ResearchGate, just because it insinuated an ill-hearted trend in those researchers, that like Zheng-Li, were deliberately twisting the evidence to fit their preconceived cover-up ideas!, while Petrovsky was straight as a man since the beginning!!! Kudos to him, and shame on the rest that twist, hide (through the Gis-NO-aid), or even "doctor" the evidence, like Shi and Holmes; so if speaking the truth gets me banned from here, feel free to seek my results of these inquires in the next root page as well, hoping there to find more peace than in FB, YT, RG, etc.: https://fdocc.neocities.org/Petrovsky--Holmes.htm

A clear-cut criticism to "cover-upper" Holmes is even raised in the main article at the top of this message, as follows:

"At first glance RaTG13 is unlikely to have evolved into SARS-CoV-2 since RaTG13 is approximately 1,200 nucleotides (3.8%) different from SARS-CoV-2. Although RaTG13 is the most closely related virus to SARS-CoV-2, this sequence difference still represents a considerable gap. In a media statement evolutionary virologist Edward Holmes has suggested this gap represents 20-50 years of evolution and others have suggested similar figures. / We agree that ordinary rates of evolution would not allow RaTG13 to evolve into SARS-CoV-2 but we also believe that conditions inside the lungs of the miners were far from ordinary."

Will Holmes... have again that information also removed????